I informed you thusly: Part 2

20 February 2023

Vaccine-induced Antibodies Don’t Confer Immunity Against Viruses that Replicate in Respiratory Mucosa – like influenza and SARS-CoV-2.

In last week’s post, I told you that masks don’t work to prevent viral respiratory illness. Not for source control, not for infection protection, not when worn in a healthcare setting by properly-trained professionals. They Just Don’t Work.

Previously, I told you that influenza vaccines don’t prevent infection with, or transmission of, influenza virus, here, and here. I also told you that COVID-19 ‘vaccines’ don’t prevent infection with, or transmission of, SARS-CoV-2 here, here, and here, and here, and I explained why here.

In a nutshell, all the injected flu vaccines and all the COVID-19 ‘vaccines’ currently on the market induce production of antibodies that travel in the blood (immunoglobulin G [IgG] and circulating immunoglobulin A [IgA]), but SARS-CoV-2 replicates in the mucous membranes that line our airways (the respiratory mucosa), not in the blood. The type of antibody that is required to stop this respiratory mucosal replication is secretory IgA (sIgA). None of the COVID-19 vaccines induce production of sIgA, and neither IgG nor circulating IgA can reach the respiratory mucosa.

Here’s the illustration of sIgA production and activity that I included in my previous article, Why COVID-19 “vaccines” will never end the pandemic which was published on 20 December 2021:

And now, a trio of crack scientists, including the recently-retired former head of the US National Institutes of Allergic and Infectious Disease, and Grand Chief Poohbah of Pandemia, Anthony S. Fauci, has published an article admitting that vaccines which only induce systemic antibodies (that is, antibodies travelling in the circulatory system) are next to useless against respiratory viruses that replicate in respiratory mucosa rather than causing systemic infection. Seriously. You can’t make this stuff up.

Here’s what the mendacious midget who fully supported the sacking and social exclusion of anyone who declined the experimental COVID-19 transfection agents, and who has forcefully pushed for mandatory influenza vaccination in healthcare workers, had to say about how poorly these agents work, and why. First, flu vaccines don’t really work:

“Until the emergence of COVID-19, influenza had for many decades been the deadliest vaccine-preventable viral respiratory disease, one for which only less than suboptimal vaccines are available… Over the years, influenza vaccines have never been able to elicit durable protective immunity against seasonal influenza virus strains, even against non-drifted strains.^4,5,6,7 Although current influenza vaccines reduce the risk of severe disease, hospitalization, and death to some degree [my note: they don’t, as I explained in my previous article Flu vaccination and faith-based medicine, which also features a choice quote from the aforementioned Tom Jefferson on the ‘rubbish’ quality of flu vaccine clinical trials] their effectiveness against clinically apparent infection is decidedly suboptimal, ranging from 14% to 60% over the past 15 influenza seasons.¹ Furthermore, the duration of vaccine-elicited immunity is measured only in months…

As of 2022, after more than 60 years of experience with influenza vaccines, very little improvement in vaccine prevention of infection has been noted. As pointed out decades ago, and still true today, the rates of effectiveness of our best approved influenza vaccines would be inadequate for licensure for most other vaccine-preventable diseases.⁷“

Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses

Let that sink in. In many jurisdictions, certain occupations, including healthcare, aged care and disability care workers, are subject to mandatory flu vaccination. In Australia, some nursing homes won’t let you visit your elderly relative if you don’t produce proof that you’ve had a flu vaccine in the current flu season. But these jabs are so ineffective that they wouldn’t pass licensure requirements if they weren’t ‘grandfathered’ by decades-old approval processes.

Next, COVID ‘vaccines’ don’t really work, either:

“As variant SARS-CoV-2 strains have emerged, deficiencies in these [COVID-19] vaccines reminiscent of influenza vaccines have become apparent. The vaccines for these two very different viruses have common characteristics: they elicit incomplete and short-lived protection against evolving virus variants that escape population immunity.^12,13,14,15 Considering that vaccine development and licensure is a long and complex process requiring years of preclinical and clinical safety and efficacy data, the limitations of influenza and SARS-CoV-2 vaccines remind us that candidate vaccines for most other respiratory viruses have to date been insufficiently protective for consideration of licensure, including candidate vaccines against RSV, a major killer of infants and the elderly,^{16,17,18,19,20,21} parainfluenzaviruses, endemic coronaviruses,²² and many other ‘common cold’ viruses that cause significant morbidity and economic loss.”

Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses

Is this the same Anthony Fauci who, in a panel discussion at the Milken Institute in 2019, appeared to be bemoaning the fact that the clinical trial process for flu vaccine development takes so long, and calling for a “disruptive” way of addressing this “problem”?

Does anyone know what Teflon Tony actually believes???

(Side note: Construction of an mRNA vaccine manufacturing facility is already underway, on the grounds of Monash University in Melbourne. What do they plan to manufacture there? “COVID-19 booster shots as well as mRNA vaccines for other respiratory viruses such as influenza and respiratory syncytial virus (RSV).” Your taxpayer dollars, hard at work again. Another mRNA vaccine ‘hub’ is being created in Brisbane.)

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Update:

As reported by Alex Berenson, Moderna’s mRNA influenza ‘vaccine’ candidate has failed to demonstrate what is coyly called ‘non-inferiority’ to conventional flu vaccines, against influenza B strains, which account for the majority of flu cases in non-elderly adults.

In addition, 70 per cent of trial participants who received the mRNA shot reported ‘solicited’ adverse reactions (i.e. adverse reactions that were explicitly asked about by trial staff) compared to 48 per cent of participants who received a conventional flu vaccine.

The Moderna shot stimulated higher antibody production against influenza A strains than the conventional flu shot, but as discussed below, serum antibody levels are not an indicator of either immunity to influenza infection or protection against severe disease. Will this throw a spanner in the works of either the Melbourne or Brisbane mRNA manufacturing facilities? I doubt it.

You can read Berenson’s report here: mRNA “vaccines” fail again.

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Next, Fauci and his co-authors explain why vaccines against flu, COVID-19, RSV and other viruses associated with colds and flu are so wretchedly ineffective. The viruses involved in diseases like measles, mumps and chickenpox are transmitted by the respiratory route, but after an initial period of rapidly-accelerating replication in the respiratory tract (which roughly correlates to their incubation period), they make their way into the bloodstream and proceed to replicate there at a spectacular rate, resulting in significant viraemia (presence of infectious viral particles in the blood). These viral particles come in contact with many different cell types in multiple compartments of the immune system, resulting in a multifaceted immune response that usually confers life-long immunity.

But cold and flu viruses are different:

“In stark contrast, the non-systemic respiratory viruses such as influenza viruses, SARS-CoV-2, and RSV tend to have significantly shorter incubation periods (Table 1) and rapid courses of viral replication. They replicate predominantly in local mucosal tissue, without causing viremia, and do not significantly encounter the systemic immune system or the full force of adaptive immune responses, which take at least 5–7 days to mature, usually well after the peak of viral replication and onward transmission to others. SARS-CoV-2 ‘RNAemia’ (circulation of viral RNA in the bloodstream, as is seen with most mucosal respiratory virus infections, as distinct from viremia, in which infectious viruses can be cultured from the blood), has been reported, and RT-PCR levels of viral RNA have been linked to severe disease,^23,24 similar to studies of influenza RNAemia.^25,26 As a result, the non-systemically replicating respiratory viruses, apparently including SARS-CoV-2,^13,14,15 tend to repeatedly re-infect people over their lifetimes without ever eliciting complete and durable protection.²⁷“

Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses

In addition, both influenza and SARS-CoV-2 mutate too rapidly for any vaccine to effectively control their spread:

“Rapid antigenic drift affects the control of annual influenza epidemics⁸ and complicates the effort to produce broadly protective, ‘universal’ influenza vaccines. The SARS-CoV-2 spike protein has shown a similar plasticity, with the emergence of multiple variants with altered antigenicity³³ that has complicated its control through current vaccination strategies.³⁴“

Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses

Wait, it gets even better:

“Taking all of these factors into account, it is not surprising that none of the predominantly mucosal respiratory viruses have ever been effectively controlled by vaccines.”

Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses

Yes, the people who accused you of being a granny-murderer, supported mandates that took away your livelihood, and egged on your exclusion from society if you didn’t accept the experimental transfection agent, are now saying, “Well, of course it doesn’t really work, dummies! Smart scientists like us never thought it would!”

Of course, the main thrust of the article is to call for the development of New! Improved! vaccines that – wait for it – mimic the body’s own processes of developing mucosal immunity. Fauci and friend discuss the development of mucosal immunity, and the fact that none of the processes induced by vaccination are involved in it, at some length:

“Many studies in humans and experimental animals, some before sIgA had been recognized,^{22,58,79,80,81} indicate that secretory mucosal immunity is generally more effective than systemic immunity in controlling mucosal respiratory viruses^18,79,82 and that tissue-resident memory T cells can be effective in rapidly responding to mucosal infection.⁸³ … Nasal sIgA is the best correlate of protection in RSV challenge studies,¹⁸ even in the absence of systemic IgA-producing B cells. Similar results are seen with other viruses, including SARS-CoV-2.^87,88,89,90 Although non-systemically replicating mucosal viruses elicit systemic effectors, including systemic IgA-producing plasma cells and in some cases high levels of serum IgA and IgG, neither circulating antibodies, plasmablasts, nor systemic B or T or T effector cells function optimally at mucosal sites. This is due in part to the dilution of transuded antibody and the fact that many such effector cells lack trafficking signals to these sites.⁸⁵“

Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses

In other words, the natural response to respiratory viruses that infect mucous membranes is to pour out sIgA to stop the virus from replicating. The elements of the immune system that ‘live’ in the circulatory system, including B cells, T cells and blood-borne antibodies, don’t make it through to the mucous membranes. Exactly as I explained in Why COVID-19 “vaccines” will never end the pandemic fourteen f#!*ing months ago.

After some discussion of the inherent difficulty of developing a vaccine that induces both mucosal immunity (which is necessary for preventing infection with, and transmission of, respiratory viruses) and immunity within the lung (which is necessary for preventing serious illness), and an admission that flu vaccines are failures at both – “current influenza vaccines are suboptimal at both preventing infection and eliciting pulmonary immunity” – Fauci and Friends drop some good news for Big Pharma:

“The implications for vaccinology are clear: preventing viral upper respiratory infection and limiting post-infection viral spread to contiguous respiratory compartments are both critical but may not be easily achieved with single vaccines.”

Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses

If one vaccine won’t do the trick, why not try two? What a bonanza for vaccine manufacturers! Will someone give Tiny Tony a seat on the board of PfizeRNAZeneca please? He’s a marketing genius!

But then comes the bombshell. After all of this hoo-hah about developing new you-beaut vaccines that simulate natural immunity, Fauci and Friends admit that they have no freakin’ idea how to tell whether any such vaccines are effective or not because they don’t fully understand how natural immunity against respiratory viruses works, and vaccine-induced ‘immunity’ simply doesn’t work the same way:

“Immune correlates of protection against mucosal respiratory viruses are incompletely understood, vary between viral strains and subtypes, with viral drift, and they exhibit inter-individual variation.
In developing next-generation vaccines, we will need to identify strong immunologic correlates of protection against each mucosal respiratory virus and agree about their relevance to public health vaccination goals.^80,101,102 Additional immune correlate studies in humans are clearly needed and should be a research priority. Following influenza infection in humans, studies have long identified serum and mucosal immunoglobulin correlates^103,104,105 and T cell immune correlates.^104,106,107 In contrast, a human influenza challenge study after vaccination with inactivated vaccines or live-attenuated influenza vaccine (LAIV), followed by LAIV challenge, was unable to find any immunologic correlates of protection.¹⁰⁸“

Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses

So yeah, those flu vaccines they’ve been pumping into everyone for decades… well, nobody really knows what the heck they’re doing. But send more money for research. They promise to use it wisely rather than – I don’t know – funnel it to a lab controlled by the Chinese military to soup up bat viruses so they can infect humans. Honestly.

In even more good news for vaccine manufacturers, the “next-generation vaccines” of Fauci’s fever dream “may need optimized formulations, higher vaccine doses, greater frequency of vaccine administration, and overcoming immune tolerance challenges.” More vaccines, more often, at higher doses, with more adjuvants. Yay for Big Pharma!

If you have read up to this point and managed not to either vomit or become apoplectic with rage, congratulations. But what I’m about to tell you might just tip you over the edge.

We already know how to raise secretory IgA production

That’s right, you don’t need a fancy-schmancy vaccine to bump up your sIgA. You just need to make sure that:

• You have optimal vitamin D status, preferably through sensible sun exposure, but you can top up with a vitamin D supplement if you live in a high latitude region;
• Your intake of vitamin A (or its carotenoid precursors, found in yellow, orange and red fruits and vegetables) is adequate;
• You have a high dietary intake of polyphenols, which are contained in fruits, vegetables, whole grains, legumes, nuts and seeds, especially those grown without nitrogen fertiliser;
• You’re proactively managing stress.

But I guess none of those things make any money for Teflon Tony and his mates, do they?

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